In every single person, under normal conditions, cells rarely but continuously become corrupted for any number of reasons (inherited mutations, environmental DNA damage, virus-related DNA damage, chemical DNA damage, or excessive replication of cells in tissues that are repaired multiple times). Usually, these cells will go through cellular suicide procedures (called apoptosis) "for the greater good" of the host. Or they begin to look different enough to be recognized and then destroyed by the immune system.
Fortuitous mutations allow the corrupted cells to resist destruction by the immune system. Some mutations shut down "suicide pathways" designed to kill off corrupted cells. Other mutations actively turn off the immune recognition of the tumor or immune response at that location.
In order for immune-resistant cells to develop into tumors, they need to be able to grow quickly. Other mutations allow the corrupt cells to grow and multiply quickly. Additional changes give tumors the ability to recruit host cells needed to provide tissue infrastructure that support and nourish the increasing tumor mass. Once this mass interferes with the functions of its host, the tumor becomes malignant and causes the disease called cancer.
Our compounds act by activating the immune system, or by reversing the inhibitory state at the tumor site. The immune system can then destroy the tumor cells, resulting in the shrinking of the tumor.
Our primary nutraceutical, MacrImmune, is a mixture of a plant protein and a plant bio-organic molecule that together shrank melanomas, sarcomas, and lymphomas in a few weeks, resulting in an over 90% cure rate, when administered orally (not injected) to mice.